Month: December 2018

Infinite Powers: How Calculus Reveals the Secrets of the Universe: Steven Strogatz

From preeminent math personality and author of The Joy of x, a brilliant and endlessly appealing explanation of calculus – how it works and why it makes our lives immeasurably better.

Without calculus, we wouldn’t have cell phones, TV, GPS, or ultrasound. We wouldn’t have tamed AIDS or discovered Neptune or figured out how to put 5,000 songs in your pocket.

Though many of us were scared away from this essential, engrossing subject in high school and college, Steven Strogatz’s brilliantly creative, down‑to‑earth history shows that calculus is not about complexity; it’s about simplicity. It harnesses an unreal number—infinity—to tackle real‑world problems, breaking them down into easier ones and then reassembling the answers into solutions that feel miraculous.

Infinite Powers recounts how calculus tantalized and thrilled its inventors, starting with its first glimmers in ancient Greece and bringing us right up to the discovery of gravitational waves (a phenomenon predicted by calculus). Strogatz reveals how this form of math rose to the challenges of each age: how to determine the area of a circle with only sand and a stick; how to explain why Mars goes “backwards” sometimes; how to make electricity with magnets; how to ensure your rocket doesn’t miss the moon; how to cure infectious diseases.

As Strogatz proves, calculus is truly the language of the universe. By unveiling the principles of that language, Infinite Powers makes us marvel at the world anew.

Source: www.amazon.com

Macroscopic dynamics and the collapse of urban traffic

Stories of mega-jams that last tens of hours or even days appear not only in fiction but also in reality. In this context, it is important to characterize the collapse of the network, defined as the transition from a characteristic travel time to orders of magnitude longer for the same distance traveled. In this multicity study, we unravel this complex phenomenon under various conditions of demand and translate it to the travel time of the individual drivers. First, we start with the current conditions, showing that there is a characteristic time τ that takes a representative group of commuters to arrive at their destinations once their maximum density has been reached. While this time differs from city to city, it can be explained by Γ, defined as the ratio of the vehicle miles traveled to the total vehicle distance the road network can support per hour. Modifying Γ can improve τ and directly inform planning and infrastructure interventions. In this study we focus on measuring the vulnerability of the system by increasing the volume of cars in the network, keeping the road capacity and the empirical spatial dynamics from origins to destinations unchanged. We identify three states of urban traffic, separated by two distinctive transitions. The first one describes the appearance of the first bottlenecks and the second one the collapse of the system. This collapse is marked by a given number of commuters in each city and it is formally characterized by a nonequilibrium phase transition.

Macroscopic dynamics and the collapse of urban traffic
Luis E. Olmos, Serdar Çolak, Sajjad Shafiei, Meead Saberi, and Marta C. González
PNAS December 11, 2018 115 (50) 12654-12661

https://doi.org/10.1073/pnas.1800474115

Source: www.pnas.org

Cancer: a complex disease

The study of complex systems and their related phenomena has become a major research venue in the recent years and it is commonly regarded as an important part of the scientific revolution developing through the 21st century. The science of complexity is concerned with the laws of operation and evolution of systems formed by many locally interacting elements that produce collective order at spatiotemporal scales larger than that of the single constitutive elements. This new thinking, that explores formally the emergence of spontaneous higher order and feedback hierarchies, has been particularly successful in the biological sciences. One particular life-threatening disease in humans, overwhelmingly common in the modern world is cancer. It is regarded as a collection of phenomena involving anomalous cell growth caused by an underlying genetic instability with the potential to spread to other parts of the human body.

In the present book, a group of well recognised specialists discuss new ideas about the disease. These authors coming from solid backgrounds in physics, mathematics, medicine, molecular and cell biology, genetics and anthropology have dedicated their time to write an authoritative free-available text published under the open access philosophy that hopefully would be in the front-line struggle against cancer, a complex disease.

 

Cancer: a complex disease
by Elena R. Alvarez-Buylla • Juan Carlos Balandran • Jose Luis Caldu-Primo • Jose Davila-Velderrain • Jennifer Enciso • Enrique Hernandez-Lemus • Lucia S. Lopez Castillo • Juan Carlos Martinez-Garcia • Nancy R. Mejia-Dominguez • Leticia R. Paiva • Rosana Pelayo • Osbaldo Resendis-Antonio • Octavio Valadez-Blanco

Source: scifunam.fisica.unam.mx

ChauffeurNet: Learning to Drive by Imitating the Best and Synthesizing the Worst

Our goal is to train a policy for autonomous driving via imitation learning that is robust enough to drive a real vehicle. We find that standard behavior cloning is insufficient for handling complex driving scenarios, even when we leverage a perception system for preprocessing the input and a controller for executing the output on the car: 30 million examples are still not enough. We propose exposing the learner to synthesized data in the form of perturbations to the expert’s driving, which creates interesting situations such as collisions and/or going off the road. Rather than purely imitating all data, we augment the imitation loss with additional losses that penalize undesirable events and encourage progress — the perturbations then provide an important signal for these losses and lead to robustness of the learned model. We show that the ChauffeurNet model can handle complex situations in simulation, and present ablation experiments that emphasize the importance of each of our proposed changes and show that the model is responding to the appropriate causal factors. Finally, we demonstrate the model driving a car in the real world.

 

ChauffeurNet: Learning to Drive by Imitating the Best and Synthesizing the Worst
Mayank Bansal, Alex Krizhevsky, Abhijit Ogale

Source: arxiv.org

Modeling Memory Effects in Activity-Driven Networks

Activity-driven networks (ADNs) have recently emerged as a powerful paradigm to study the temporal evolution of stochastic networked systems. All the information on the time-varying nature of the system is encapsulated into a constant activity parameter, which represents the propensity to generate connections. This formulation has enabled the scientific community to perform effective analytical studies on temporal networks. However, the hypothesis that the whole dynamics of the system is summarized by constant parameters might be excessively restrictive. Empirical studies suggest that activity evolves in time, intertwined with the system evolution, causing burstiness and clustering phenomena. In this paper, we propose a novel model for temporal networks, in which a self-excitement mechanism governs the temporal evolution of the activity, linking it to the evolution of the networked system. We investigate the effect of self-excitement on the epidemic inception by comparing the epidemic threshold of a Susceptible–Infected–Susceptible model in the presence and in the absence of the self-excitement mechanism. Our results suggest that the temporal nature of the activity favors the epidemic inception. Hence, neglecting self-excitement mechanisms might lead to harmful underestimation of the risk of an epidemic outbreak. Extensive numerical simulations are presented to support and extend our analysis, exploring parameter heterogeneities and noise, transient dynamics, and immunization processes. Our results constitute a first, necessary step toward a theory of ADNs that accounts for memory effects in the network evolution.

Modeling Memory Effects in Activity-Driven Networks
Lorenzo Zino, Alessandro Rizzo, and Maurizio Porfiri

SIAM J. Appl. Dyn. Syst., 17(4), 2830–2854. (25 pages)

Source: epubs.siam.org